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Current Topics in Medicinal Chemistry, Volume 4, No. 1, 2004

 

Contents

 

Opioids: Receptors, Analogs, Biological Functions and Therapeutic Applications

Guest Editor: Tomasz Janecki

 

Opioid Receptors and their Ligands Pp.1-17

Anna Janecka, Jakub Fichna and Tomasz Janecki

[Abstract]

 

New Trends in the Development of Opioid Peptide Analogues as Advanced Remedies for Pain Relief Pp.19-38

Luca Gentilucci

[Abstract]

 

Endogenous Opioids and Addiction to Alcohol and other Drugs of Abuse Pp.39-50

Christina Gianoulakis

[Abstract]

 

Expression of Opioid Receptors During Peripheral Inflammation Pp.51-61

Olga Pol and Margarita M. Puig

[Abstract]

 

Neuropeptides and Gastric Mucosal Homeostasis Pp.63-73

Gyires, K.

[Abstract]

 

Substance P: Structure, Function, and Therapeutics Pp.75-103

Prasanna Datar, Sudha Srivastava, Evans Coutinho and Girjesh Govil

[Abstract]

 

Recent Advances in the Investigation of the Bioactive Conformation of Peptides Active at the µ-opioid Receptor. Conformational Analysis of Endomorphins Pp.105-121

Luca Gentilucci and Alessandra Tolomelli

[Abstract]

 

Use of NMR and Fluorescence Spectroscopy as well as Theoretical Conformational Analysis in Conformation -activity Studies of Cyclic Enkephalin Analogues Pp.123-133

Joanna Malicka, Cezary Czaplewski, Malgorzata Groth, Wieslaw Wiczk, Stanislaw Oldziej, Leszek Lankiewicz, Jerzy Ciarkowski, Adam Liwo

[Abstract]

 

Conformational Analysis of Opioid Peptides in the Solid States and the Membrane Environments by NMR Spectroscopy Pp.135-145

Akira Naito and Katsuyuki Nishimura

[Abstract]

 

Antagonism in Opioid Peptides: the Role of Conformation Pp.147-157

Severo Salvadori and Piero A. Temussi

[Abstract]

 

Abstracts

 

[Back to top] Opioid Receptors and their Ligands

Anna Janecka, Jakub Fichna and Tomasz Janecki

 

This review gives a historical perspective, summarizing approximately 25 years of research on opioids. The “typical” opioid peptides produced in the brain, “atypical” opioids encrypted in milk protein or hemoglobin sequences, and extremely potent and selective opioidsof amphibian origin are described. The main focus is on the structure-activity relationship studies of peptide ligands for three main opioid receptor types (m, d, k), their selectivities and pharmacological activities in vitro. Chemical modifications that led to obtaining potent and selective agonists and antagonists for these receptors are discussed.

 

[Back to top] New Trends in the Development of Opioid Peptide Analogues as Advanced Remedies for Pain Relief

Luca Gentilucci

 

The search for new peptides to be used as analgesics in place of morphine has been mainly directed to develop peptide analogues or peptidomimetics having higher biological stability and receptor selectivity. Indeed, most of the alkaloid opioid counterindications are due to the scarce stability and the contemporary activation of different receptor types. However, the development of several extremely stable and selective peptide ligands for the different opioid eceptors, and the recent discovery of the m-receptor selective endomorphins, rendered this search less fundamental. In recent years, other opioid peptide properties have been investigated in the search for new pharmacological tools. The utility of a drug depends on its ability to reacappropriate receptors at the target tissue and to remain metabolically stable in order to produce the desired effect. This review deals with the recent investigations on peptide bioavailability, in particular barrier penetration and resistance against enzymatic degradation; with the development of peptides having activity at different receptors; with chimeric peptides, with propeptides, and with non-conventional peptides, lacking basic pharmacophoric features.

 

[Back to top] Endogenous Opioids and Addiction to Alcohol and other Drugs of Abuse

Christina Gianoulakis

 

There is significant experimental evidence implicating the endogenous opioid system (opioid peptides and opioid receptors) with the processes of reward and reinforcement. Indeed, many behaviors associated with reward and reinforcement, for example feeding behavior, are controlled by distinct components of the endogenous opioid system located in relevant brain regions. It has also been shown that regardless of their initial site of action many drugs of abuse, such as morphine, nicotine, cocaine, alcohol and amphetamines, induce an increase in the extracellular concentration of dopamine in the nucleus accumbens. This increased secretion of dopamine in the nucleus accumbens seems to be a common effect of many drugs of abuse, and it was proposed that may mediate their rewarding and reinforcing properties. Furthermore, activation of m opioid receptors in the ventral tegmental area, or of m and d opioid receptors in the nucleus accumbens enhances the extracellular concentration of dopamine in the nucleus accumbens. Thus, stimulation of the activity of distinct components of the endogenous opioid system either by opioid or by other drugs of abuse, may mediate some of their reinforcing effects. In this review article, a brief description of the endogenous opioid system and its implication in the processes of reward and reinforcement of opioid and other drugs of abuse will be presented. Furthermore, the use of opioid antagonists in the treatment of drug addiction will be discussed. Special emphasis will be given to ethanol addiction, the drug mainly studied in my laboratory.

 

[Back to top] Expression of Opioid Receptors During Peripheral Inflammation

Olga Pol and Margarita M. Puig

 

Opioid receptors (OR) and their mRNA are present in the central and peripheral nervous system of mammals. In this review we examine the behavioral effects of opioids and the expression of their receptors during peripheral inflammation in two experimental models: the rat paw and the mouse intestine. Inflammation increased the antinociceptive (paw ) and the inhibitory effects of opioids in the gut (transit, permeability and plasma extravasation) by inte ra ction with OR loca ted a t peripheral sites. Based on agonist efficacy, m > d >> k-OR mediate the antinociceptive and antitransit effects of opioids during inflammation. Intestinal permeability is modulate d by d = m >> k-O R, while k > d >> m-O R are involved in the inhibition of plasma extravasation. Intestinal inflammation increased the transcription of m and d-OR (but not k) genes in the gut, thus explaining the enhanced antitransit and antisecretory effects of m and d-OR agonists; however, the increased inhibitory effects of k-OR agonists on plasma extravasation could result from post-transcriptional regulation of the receptor. Similarly, the increased expression of peripheral m-OR observed in the rat paw during inflammation, occurs at post-transcriptional levels and is related to an increased axonal transport from the dorsal root ganglia to peripheral terminals. The sites and mechanisms implicated in the increased transcription of m and d-OR during intestinal inflammation are under investigation.

 

[Back to top] Neuropeptides and Gastric Mucosal Homeostasis

Gyires, K.

 

The role of central nervous system (CNS) in regulation of gastric function has long been known. The dorsal vagal complex (DVC) has an important role in regulation of gastric mucosal integrity; it is involved both in mucosal protection and in ulcer formation. Neuropeptides have been identified in DVC, the origin of these peptides are both intrinsic and extrinsic. Neuropeptides are localized also in the periphery, in afferent neurons. The afferent neurons also have efferent-like function in the gastroinetestinal tract, and neuropeptides released from the peripheral nerve endings of primary afferent neurons can induce gastric mucosal protection. Centrally and /or peripherally injected neuropeptides, such as amylin, adrenomedullin, bombesin, cholecystokinin, neurotensin, opioid peptides, thyreotropin releasing hormone and vasoactive intestinal peptide, influence both the acid secretion and the gastric mucosal lesions induced by different ulcerogens. The centrally induced gastroprotective effect of neuropeptides may be partly due to a vagal dependent increase of gastric mucosal resistance to injury; activation of vagal cholinergic pathway is resulted in stimulation of the  release of mucosal prostaglandin and nitric oxide. Furthermore, release of sensory neuropeptides (calcitonin gene-relatedpeptide, tachykinins) from capsaicin sensitive afferent fibers are also involved in the centrally induced gastroprotective effect of neuropeptides.

 

[Back to top] Substance P: Structure, Function, and Therapeutics

Prasanna Datar, Sudha Srivastava, Evans Coutinho and Girjesh Govil

 

Extensive efforts since 1931, on the structural determination of the mammalian tachykinin SP by NMR, CD and IR have turned out to be inconclusive. Studies are now being concentrated on the structural properties and characteristics of various NK receptors (NK1, NK2 and NK3) with the help of genetics, cloning, receptor engineering, mutagenesis and modeling. This knowledge is now being fruitfully used in the development of non-peptide NK1 receptor antagonists that essentially block the pharmacological effects of SP. It is now being realized that the simultaneous blockade of two or more receptors gives promising results in emesis, depression and pulmonary obstructive diseases. In addition to the synthetic compounds, the discovery of antagonists from natural origin has added a great value to this field. In this review we have made an attempt to present the structural characteristicsof SP, its analogs and antagonists, the structural characteristics of the NK receptor, and structure activity relationships that have helped to improve the therapeutic utilities of SP antagonists.

 

[Back to top] Recent Advances in the Investigation of the Bioactive Conformation of Peptides Active at the m-opioid Receptor. Conformational Analysis of Endomorphins

Luca Gentilucci and Alessandra Tolomelli

 

Despite of the recent advances in the structural investigation of complex molecules, the comprehension of the 3D features responsible for the interaction between opioid peptides and m- opioid receptors still remains an elusive task. This has to be attributed to the intrinsic nature of opioid peptides, which can assume a number of different conformations of similar energy, and to the flexibility of the receptorial cavity, which can modify its inner shape to host different ligands. Due to this inherent mobility of the ligand-receptor system, massive efforts devoted to the definition of a rigid bioactive conformation to be used as a template for the design of new pharmacologically active compounds might be overstressed. The future goal might be the design of peptide or nonpeptide ligands capable of maximizing specific hydrophobic interactions. This review covers the recent opinions emerged on the nature of the ligand-receptor interaction, and the development of suitable models for the determination of the bioactive conformation of peptide ligands active towards m-opioid receptors.

 

[Back to top] Use of NMR and Fluorescence Spectroscopy as well as Theoretical Conformational Analysis in Conformation activity Studies of Cyclic Enkephalin Analogues

Joanna Malicka, Cezary Czaplewski, Malgorzata Groth, Wieslaw Wiczk, Stanislaw Oldziej, Leszek Lankiewicz, Jerzy Ciarkowski, Adam Liwo

 

In this review the conformational studies of natural enkephalins (H-Tyr-Gly-Gly-Phe- Met-OH; the [Met5]enkephalin and H-Tyr-Gly-Gly-Phe-Leu-OH; the [Leu5]enkephalin), their acyclic and cyclic analogues, including those carried out in our laboratory, performed by experimental and theoretical methods and their combination, are described. Emphasis is given on the role of conformational constraints introduced by cyclization on activity at the m and d opioid receptors. Comparison of the conformations of cyclic enkephalin analogues with high d-receptor activity with those of potent rigid non-peptide d-receptor agonists indicates that the proximity of the aromatic side chains in positions 1 and 4 as well as the N-terminal amino group is desirable for the activity at the d opioid receptors; early conformational studies also suggested that spatial separation of the aromatic side chains and rigidity of the cyclic backbone is desirable for m-receptor activity. The results of our recent conformational studies performed with the use of fluorescence and NMR spectroscopy as well as theoretical calculations indicate, however, that these structural features are not necessary for activity at the m opioid receptors. Methods applied to the determination of the conformation of flexible peptides, such as Nuclear Magnetic Resonance (NMR), fluorescence spectroscopy, and theoretical conformational analysis are also discussed briefly.

 

[Back to top] Conformational Analysis of Opioid Peptides in the Solid States and the Membrane Environments by NMR Spectroscopy

Akira Naito and Katsuyuki Nishimura

 

Determination of conformations and structures of opioid peptides in the membrane environments is an essential step to understand the action of the peptide to the specialized receptors. This information not only gains insight into the structure-function relationship of opioid peptide but also gives proper guidelines to design a new drug to have same neuroendocrine functions. This review provides the structural studies of three types of opioid peptide families such as enkephalin, b-endorphin and dynorphin in the solid states and the membrane environments. The structures of enkephalins show that they take b-bend, extended and double b-bend structures in the crystals. Moreover, enkephalin molecules take a variety of structures in the crystals and are easily converted to the other structures with slightly different torsion angles. On the other hand, b-bend structures are mostly seen in the membrane environments. Membrane bound structure of dynorphin shows that the N-terminus forms a-helixal structure and is inserted into the membrane with the helical axis almost perpendicular to the membrane surface. It is discussed that the helical region of the extracellular loop II of the κ- opioid receptor may interact with the helical region of dynorphin with a high affinity in the membrane environments. b- endorphin takes a-helical structure at N-terminus and the central regions and the rest of regions take unordered structure when they bind to the membrane. Since the membrane bound structures of opioid peptides differ from those of the solution states, membrane association is an important process for exerting the affinity and the selectivity to the specific opioid receptors.

 

[Back to top] Antagonism in Opioid Peptides: the Role of Conformation

Severo Salvadori and Piero A. Temussi

 

The availability of new, highly selective antagonists, in the field of opioid peptides and of other pain peptides, is important both for a better understanding of the interaction of the receptors with their ligands and for their practical relevance.

 

The design of antagonists is not obvious even when the essential features of agonists are well known. In this review we have examined the main aspects of the problem using, as leading criteria two theoretical models of antagonism and the subdivision of opioid peptides into two functional domains.

 

The main causes of antagonism have been integrated in two very general models: one, referred to as the participation model, attributes antagonism to the lack, with respect to the parent agonist, of an essential group, whereas another model, attributes antagonism to the misfit of the molecule inside the receptor.

 

The second criterion is the division of the structure of peptide hormones, originally put forward by Robert Schwyzer, in two functional domains, the message domain, which is responsible of the larger part of the binding affinity of opioid agonists, and an address domain, which dictates most of the peptide specificity.

 

The most significant achievements in the design of opioid antagonists are classified according to the relative importance of chemical constitution, conformation and chirality.