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Mini-Reviews in Medicinal Chemistry, Volume 4, No. 6, 2004

 

Contents

 

Arachidonic Acid Cascade Modulators: The Cyclooxygenase Pathway

Executive Editors: Jean-Michel Dogne / Xavier de Leval

 

First and Second Generations of COX-2 Selective Inhibitors Pp. 597-601

Xavier de Leval, Fabien Julemont, Valerie Benoit, Michel Frederich, Bernard Pirotte and Jean-Michel Dogne

[Abstract]

 

Structural Approach for COX-2 Inhibition Pp. 603-615

C. Michaux and C. Charlier

[Abstract]

 

The Second Generation of COX-2 Inhibitors: Clinical Pharmacological Point of View Pp. 617-624

D.O. Stichtenoth

[Abstract]

 

COX-2 Selective Inhibitors, Carbonic Anhydrase Inhibition and Anticancer Properties of Sulfonamides Belonging to This Class of Pharmacological Agents Pp. 625-632

Claudiu T. Supuran, Angela Casini, Antonio Mastrolorenzo and Andrea Scozzafava

[Abstract]

 

Recent Development in the Field of Dual COX / 5-LOX Inhibitors Pp. 633-638

Fabien Julemont, Jean-Michel Dogne, Bernard Pirotte and Xavier de Leval

[Abstract]

 

Advance in Understanding the Biosynthesis of Prostacyclin and Thromboxane A2 in the Endoplasmic Reticulum Membrane via the Cyclooxygenase Pathway Pp. 639-647

Ke-He Ruan

[Abstract]

 

New Developments on Thromboxane Modulators Pp. 649-657

Jean-Michel Dogne, Julien Hanson, Xavier de Leval, Bernard Masereel, Philippe Kolh and Bernard Pirotte

[Abstract]

 

Modulation of the Arachidonic Cascade with ω3 Fatty Acids or Analogues: Potential Therapeutic Benefits Pp. 659-668

Isabelle Roland, Xavier de Leval, Brigitte Evrard, Bernard Pirotte, Jean-Michel Dogne and Luc Delattre

[Abstract]

 

“Dipeptoids”: From the Chemical Structure of the Endogenous Peptide to the Design of Peptidomimetics Pp. 669-680

Mercedes Martin-Martinez, Rosario Patino-Molina, M. Teresa Garcia-Lopeza and Rosario Gonzalez-Muniza

[Abstract]

 

Predicting Synthetic Accessibility: Application in Drug Discovery and Development Pp. 681-692

J.C. Baber and M. Feher

[Abstract]

 

The Development of Inhibitors of Heparanase, a Key Enzyme Involved in Tumour Metastasis, Angiogenesis and Inflammation Pp. 693-702

Vito Ferro, Edward Hammond and Jon K. Fairweather

[Abstract]

 

Abstracts

 

[Back to top] First and Second Generations of COX-2 Selective Inhibitors

Xavier de Leval, Fabien Julemont, Valerie Benoit, Michel Frederich, Bernard Pirotte and Jean-Michel Dogne

 

The identification and characterization of the inducible form of cyclooxygenases (COX-2) stimulated the investigations to develop efficient, non-steroidal anti-inflammatory drugs (NSAIDs) with reduced side effects (essentially gastro-intestinal toxicity) compared to classical NSAIDs. This review focuses on the chemical and pharmacological properties (pre-clinical data) of marketed COX-2 inhibitors.

 

[Back to top] Structural Approach for COX-2 Inhibition

C. Michaux and C. Charlier

 

The design of selective COX-2 inhibitors is a new approach to obtain potent, anti-inflammatory drugs but with less side effects. Several families of such inhibitors were reported in literature. In this review, the drug design processes used to understand their binding mode and the origin of selectivity of these compounds are described.

 

[Back to top] The Second Generation of COX-2 Inhibitors: Clinical Pharmacological Point of View

D.O. Stichtenoth

 

Valdecoxib, parecoxib, etoricoxib and lumiracoxib represent the second generation of selective COX-2 inhibitors. In comparison to the first generation, they show an at least equivalent efficacy in the treatment of pain and inflammation. However, the postulated gain of safety is yet difficult to determine and seems to be, if any, small.

 

[Back to top] COX-2 Selective Inhibitors, Carbonic Anhydrase Inhibition and Anticancer Properties of Sulfonamides Belonging to This Class of Pharmacological Agents

Claudiu T. Supuran, Angela Casini, Antonio Mastrolorenzo and Andrea Scozzafava

 

The sulfonamides constitute an important class of drugs, with several types of pharmacological agents possessing antibacterial, anti-carbonic anhydrase, diuretic, hypoglycaemic, antithyroid, protease inhibitory and anticancer activity among others. A recently developed class of pharmacological agents incorporating primary sulfamoyl moieties in their molecule is constituted by the COX-2 selective inhibitors, with at least two clinically used drugs, celecoxib and valdecoxib. Another drug of this class, rofecoxib, does not contain sulfonamide moieties, but the isosteric and isoelectronic methylsulfone group. It was recently shown that the sulfonamide COX-2 selective inhibitors (but not the methylsulfone ones) also act as nanomolar inhibitors of several isozymes of the metallo-enzyme carbonic anhydrase (CA), some of which are strongly involved in tumourigenesis. In consequence, the potent anticancer effects of the sulfonamide COX-2 selective inhibitors and the much weaker such effects of rofecoxib, reported ultimately by many researchers, may be explained by the contribution of CA inhibition to such processes in addition to COX-2 inhibition.

 

[Back to top] Recent Development in the Field of Dual COX / 5-LOX Inhibitors

Fabien Julemont, Jean-Michel Dogne, Bernard Pirotte and Xavier de Leval

 

Cyclooxygenases and lipoxygenase are key enzymes in the arachidonic acid metabolism. Dual inhibitors are drugs able to block both the COX and the 5-LOX metabolic pathways. Compared to COX or LOX pathways single inhibitors, dual inhibitors present at least two major advantages. First, dual inhibitors, by acting on the two major arachidonic acid metabolic pathways, possess a wide range of anti-inflammatory activities. Secondly, dual inhibitors appear to be almost exempt from gastric toxicity, which is the most troublesome side effect of non-selective COX inhibitors.

 

[Back to top] Advance in Understanding the Biosynthesis of Prostacyclin and Thromboxane A2 in the Endoplasmic Reticulum Membrane via the Cyclooxygenase Pathway

Ke-He Ruan

 

Recent advances in topological and structural characterization of the prostacyclin (PGI2) and thromboxane A2 (TXA2) synthases have led to the understanding of the biosynthesis of PGI2 and TXA2 at a structural level. This mini-review focuses on the molecular mechanism of the isomerization of the prostaglandin H2 to PGI2 and TXA2 by their synthases in the endoplasmic reticulum (ER) membrane coordinated with cyclooxygenase-1 or -2. This review summarizes the evidences in which the biosynthesis of PGI2 and TXA2 are influenced/modulated by the membrane anchor residues of the synthases and the ER membrane itself, and provides the structural basis for engineering the synthases for the next generation of gene therapy and drug designs targeting the specific synthases.

 

[Back to top] New Developments on Thromboxane Modulators

Jean-Michel Dogne, Julien Hanson, Xavier de Leval, Bernard Masereel, Philippe Kolh and Bernard Pirotte

 

Thromboxane A2 (TXA2) is a labile product formed from arachidonic acid by cyclooxygenase. The pathogenesis of numerous cardiovascular, pulmonary, and thromboembolic diseases can be related to this metabolite. Therefore, TXA2 modulators have been developed for 20 years. This review will highlight the evolution in the field of TXA2 modulators.

 

[Back to top] Modulation of the Arachidonic Cascade with ω3 Fatty Acids or Analogues: Potential Therapeutic Benefits

Isabelle Roland, Xavier de Leval, Brigitte Evrard, Bernard Pirotte, Jean-Michel Dogne and Luc Delattre

 

Increasing interest in the role of ω3 fatty acids has arisen in these latest years since evidence of their implication in the cardioprotective fish based diet of the Inuit has been demonstrated. Furthermore, several in vitro, in vivo and epidemiological studies support the benefit of this fatty acids intake in various pathological states such as in the cardiovascular, cancer, inflammation, psychiatric, paediatric, pulmonary, dermatological and ophthalmologic fields. This review will focus on metabolism and pharmacological implication of ω3 fatty acids intake as well as its interest in the prevention or treatment of the above-mentioned pathologies.

 

[Back to top] “Dipeptoids”: From the Chemical Structure of the Endogenous Peptide to the Design of Peptidomimetics

Mercedes Martin-Martinez, Rosario Patino-Molina, M. Teresa Garcia-Lopeza and Rosario Gonzalez-Muniza

 

The present review details the rational multi-step process followed for the discovery of a family of non-peptide CCK receptor ligands (“dipeptoids”), starting from the structure of the endogenous peptide, CCK8. Emphasis will be made on the N- and C-terminal modifications, on the singular effects of the stereochemical changes and the incorporation of conformational constraints into the structure of “dipeptoids”, and on the modifications directed to improve the pharmacological profile of these compounds to afford valuable clinical candidates.

 

[Back to top] Predicting Synthetic Accessibility: Application in Drug Discovery and Development

J.C. Baber and M. Feher

 

The estimation and use of synthetic accessibility in the drug discovery process is discussed. A distinction is drawn between synthetic feasibility and accessibility and the practical uses of an accessibility score are examined. Various techniques used in the estimation of accessibility are described and their utility and potential accuracy compared.

 

[Back to top] The Development of Inhibitors of Heparanase, a Key Enzyme Involved in Tumour Metastasis, Angiogenesis and Inflammation

Vito Ferro, Edward Hammond and Jon K. Fairweather

 

Heparanase is an endo-β-glucuronidase that degrades the glycosaminoglycan heparan sulfate, a major component of the extracellular matrix and basement membranes, and has been implicated in such processes as inflammation, angiogenesis and metastasis. The identification of inhibitors of heparanase is an attractive approach towards developing new therapeutics for metastatic tumours and chronic inflammatory diseases. This review focuses on heparanase inhibitors that have been isolated or synthesised to date. More recent developments in the understanding of heparanase structure and function that may ultimately aid in the future design of inhibitors with improved potency and specificity, are also discussed.